What is the primary therapeutic effect of low-dose aspirin for a client with coronary artery disease?

The primary therapeutic effect of low-dose aspirin for a client with coronary artery disease is its antiplatelet effect. Low-dose aspirin works by inhibiting the function of platelets, which are blood cells that help with clotting. This anticoagulant property is crucial for individuals with coronary artery disease, as it helps to prevent the formation of clots that could obstruct the arteries and lead to serious events such as heart attacks or strokes.

By reducing the ability of platelets to aggregate, low-dose aspirin improves blood flow and lowers the risk of cardiovascular complications. This particular action is especially significant as maintaining open and clear coronary arteries is essential for optimal heart function and overall health in patients with existing heart conditions.

The other options do not apply to the primary therapeutic use of low-dose aspirin in this context, as analgesic refers to pain relief, antidepressant refers to mood enhancement, and antihypertensive relates to lowering blood pressure, none of which are the main purpose of administering low-dose aspirin for coronary artery disease.